Symptomatic hypocalcemia in primary hyperaldosteronism: A case report

Sachin G. Pai, K. N. Shivashankara, V. Pandit, S. Sheshadri

Research output: Contribution to journalArticle

Abstract

The metabolic alterations caused by hyperaldosteronism are being increasingly recognized and have generated considerable interest among the medical fraternity. Hyperaldosteronism is suspected to have a pivotal role in the patho-physiology of congestive cardiac failure where it has been studied extensively. But its effects on calcium metabolism, parathyroid metabolism and renal handling of calcium are less well described. Recent experimental models have shed light into the roles played by previously unknown mechanisms in causing these metabolic alterations. We hereby report a case of primary hyperaldosteronism due to adrenal adenoma (Conn's syndrome) who presented with a myriad of clinical features including symptomatic hypocalcemia, significant weight loss along with uncontrolled hypertension for a prolonged period before eventually detected to have primary hyperaldosteronism. Surgical removal of the causative tumor resulted in prompt disappearance of all the symptoms and signs and regain of lost weight.

Original languageEnglish
Pages (from-to)1220-1223
Number of pages4
JournalJournal of Korean Medical Science
Volume24
Issue number6
DOIs
Publication statusPublished - 01-12-2009

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Hyperaldosteronism
Hypocalcemia
Calcium
Adenoma
Signs and Symptoms
Weight Loss
Theoretical Models
Heart Failure
Hypertension
Kidney
Weights and Measures
Neoplasms

All Science Journal Classification (ASJC) codes

  • Medicine(all)

Cite this

Pai, Sachin G. ; Shivashankara, K. N. ; Pandit, V. ; Sheshadri, S. / Symptomatic hypocalcemia in primary hyperaldosteronism : A case report. In: Journal of Korean Medical Science. 2009 ; Vol. 24, No. 6. pp. 1220-1223.
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Symptomatic hypocalcemia in primary hyperaldosteronism : A case report. / Pai, Sachin G.; Shivashankara, K. N.; Pandit, V.; Sheshadri, S.

In: Journal of Korean Medical Science, Vol. 24, No. 6, 01.12.2009, p. 1220-1223.

Research output: Contribution to journalArticle

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